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Nogo-A สมาชิกของตระกูล reticulon ผลิตโดย<br>oligodendrocytes [8] และเป็นสารยับยั้งที่มีศักยภาพของการเปลี่ยนแปลงตามแนวแกน Nogo-A โต้ตอบกับตัวรับ Nogo-66 และยับยั้ง<br>neurite outgrowth หลังจากได้รับบาดเจ็บ CNS [9] แอนติบอดี monoclonal (IN-1) กับ Nogo-A ถูกสร้างขึ้นหลายปีก่อน<br>รหัสของ Nogo-A [10] เพราะ Nogo-A เล่น<br>บทบาทสําคัญหลังจากการบาดเจ็บของ CNS การศึกษาจํานวนมากมี<br>มีวัตถุประสงค์เพื่อปิดกั้นการทํางานของมันโดยการแช่แอนติบอดีกับ<br>Nogo-A หรือตัวรับ Nogo-66 หลังจากโรคหลอดเลือดสมอง
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